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Finasteride binds to type II 5 α reductase which is found mostly in the prostate and genital tract. Dutasteride binds to both type 1 (found mostly in extraprostatic tissues) and type II 5 α reductase. dihydrotestosterone is vital for physiologic erections as it activates nitric oxide synthase and increases blood flow in cavernosal tissue. patients on finasteride are more likely to have a loss of libido, ejaculatory disorder, and erectile dysfunction. Neuropsychiatric medications accounted for the second common cause of erectile dysfunction. The antipsychotic paliperidone and the selective serotonin reuptake inhibitors citalopram and sertraline were found to have elevated high rate of erectile dysfunction. In addition, there are many neuropsychiatric medications with high frequencies of erectile dysfunction including escitalopram, quetiapine, olanzapine, fluoxetine, venlafaxine, risperidone, aripiprazole, gabapentin, pregabalin, and oxycodone. 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For example, the antipsychotics that block α1 adrenergic receptors may cause retrograde ejaculation but not erectile dysfunction. Benzodiazepines may cause sexual dysfunction from potentiation of GABA in the reticular and limbic system and by affecting the serotonin and dopaminergic pathways. Cardiological medications accounted some of rug induced erectile dysfunction. Beta blockers have been shown to be associated with ED, likely secondary to suppression of central nervous system sympathetic outflow. Non–cardioselective beta antagonists like propranolol have a higher incidence of erectile dysfunction than cardioselective beta antagonists which avoid beta 2 inhibition resulting in vasoconstriction of the corpora cavernosa. Nebivolol has the greatest selectivity for beta 1 receptors as well as endothelial nitric oxide vasodilatory effects, and has been shown to have a positive effect on erections. thiazide diuretics can also induce erectile dysfunction. In contrast, there is less evidence that loop diuretics such as furosemide cause ED, which should be considered by physicians treating hypertensive men. Some data suggests that ejaculatory dysfunction with calcium channel blockers (from decreased bulbocavernosus muscle force). Angiotensin-converting enzyme inhibitors and angiotensin receptor blockers are common antihypertensives that may actually improve erectile function. Angiotensin converting enzyme inhibitors prevent the formation of angiotensin II while angiotensin receptor blockers block the peripheral vasoconstriction caused by angiotensin II. Without angiotensin IIs effects, there is less penile vasoconstriction and collagen remodeling of the corpora cavernosa. In most studies, ACE inhibitors and ARBs have not been associated with ED and some studies even showed a beneficial effect. Medicines used for Dermatology and Immunology can also cause erectile dysfunction. 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