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Use(s) of this information is entirely the responsibility of those who choose to apply this information for their personal health and/or well being. This information is not intended as a prescription, prognosis or diagnosis for any disease or illness and should not be used as a replacement for any medical treatment you may currently be undergoing. The information provided is solely the opinion of the individual(s) present in this video. Application of the information provided without supervision of a licensed doctor is done at the individuals own risk. #holistichealth #dandelionroot #beyonceEvaluating Keto Boost As Featured On Shark TankCan you eat starches while dieting? 🍠🥔 Dr. Mike Israetel shares the best starchy foods to fuel your weight loss journey without derailing progress. 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Grassroots Labs | Save 30-70% off lab testing you need when visiting the doctor or managing your health. https://bit.ly/4fIeazG Follow Us: Facebook: https://bit.ly/3W3qTpB Instagram: https://www.instagram.com/grassroots_labs/ X: https://x.com/grassrootslabs?lang=enUnlimited Steaks At Prime Steakhouse Keto Diet All Meat Carnivore Diet Semaglutide, a medication originally developed to treat type 2 diabetes, has recently gained popularity for its off-label use as a weight loss aid. While the FDA has approved a higher dose of semaglutide specifically for weight loss, many people are unsure of how much semaglutide to take for weight loss and what the appropriate dosage is. In this article, we will provide a comprehensive dosage guide for semaglutide for weight loss, including recommended doses, potential side effects, and tips for safe and effective use. Recommended Dose The recommended dose of semaglutide for weight loss is 2.4 mg injected once weekly. 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When starting semaglutide for weight loss, your healthcare provider may recommend starting at a lower dose and gradually increasing to the full 2.4 mg dose over several weeks. This can help to minimize potential side effects, such as nausea, vomiting, or diarrhea, which may occur when first starting semaglutide or when increasing the dose. It is important to talk to your healthcare provider if you experience any side effects while taking semaglutide for weight loss. Side Effects While semaglutide is generally well-tolerated, some individuals may experience side effects when taking this medication for weight loss. Common side effects of semaglutide may include nausea, vomiting, diarrhea, constipation, or abdominal pain. These side effects are usually mild to moderate in severity and may improve over time as your body adjusts to the medication. In rare cases, semaglutide may cause more serious side effects, such as pancreatitis or gallbladder problems. 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I'm Ileana Piña, professor of medicine at Jefferson University Hospital in Philadelphia. I'm the chief of quality. This is my blog. We have been hearing a lot of conversation about obesity. It's coming out of our endocrine division and our weight management division. And the patients are asking for things like bariatric surgery and maybe a holistic program of weight loss. A lot of the primary care doctors are starting to recommend glucagon-like peptide-1 (GLP-1) receptor agonists for those patients. But we deal with patients with heart failure with preserved ejection fraction (HFpEF), and a lot of our phenotypes are patients with obesity who happen to have hypertension and/or diabetes, and they are difficult to manage. Inflammation, Obesity, and HFpEF Piña: For today's conversation, there is nobody better than Dr Ambarish Pandey, who has a HFpEF program at the University of Texas Southwestern. Ambarish, welcome. Give me your thoughts overall about the link between obesity, inflammation, and HFpEF. Ambarish Pandey, MD: Thank you, Dr Piña. I'm excited to be on your blog and I'm glad that we are discussing such an important topic. The burden of heart failure, particularly HFpEF, has been increasing and is not just affecting older individuals. We see more younger folks with obesity in our HFpEF clinic coming in with symptoms of shortness of breath and fluid buildup. Our understanding of HFpEF has evolved quite a bit over the past two or three decades. Initially, it was thought to be a disease of largely impaired myocardial relaxation. And then Walter Paulus was one of the pioneers who proposed the concept of inflammation leading to HFpEF, and obesity being one of the important drivers of the inflammation that leads to the development of HFpEF. Now our understanding of HFpEF has evolved to it being a multisystem disorder that leads to dysfunction in different organ systems. One of the cardinal features that drives this dysfunction is upregulation of inflammatory pathways, increased inflammatory burden — and a lot of that is driven by obesity. Obesity is one of the most important modifiable risk factors for HFpEF. Piña: It is so common. In my clinic the other day, probably 3 out of 4 patients were obese. Pandey: Up to 75% of patients that we see in our program are over the 30 BMI cut-off, and a lot of the literature that has come from US HFpEF cohorts shows a very high average BMI in these patients. Piña: When we talk about inflammation, what are the chemical pathways of inflammation? For example, a long time ago, we tried to block TNF-alpha and it didn't quite work out. So, what pathways are we talking about? Pandey: I think a lot more work needs to be done to really understand the key drivers of inflammation and what pathways are being upregulated. As we know, inflammation is most commonly identified by elevation in levels of C-reactive protein (CRP), which serves as a marker of increased inflammation, but the upstream upregulation in IL-1– and IL-6–mediated pathways has been implicated in inflammation that drives development of diseases like HFpEF. There is an ongoing trial of ziltivekimab, which is an IL-1 inhibitor in patients with HFpEF. That's the HERMES trial led by Paul Ridker, one of the pioneers of inflammatory therapies in heart disease. We are starting to understand more about the key molecular pathways that are driving it. And now we're starting to target them, with the hope that we can actually halt the progression of HFpEF and improve the symptom burden as well as the risk for adverse outcomes in these patients. Are GLP-1 Inhibitors Anti-inflammatory? Piña: Do you think that the GLP-1s, or even now the SGLT2 inhibitors, are anti-inflammatory since we really don't know all the pathways? Pandey: I do think that GLP-1s, by virtue of causing weight loss and reduction in visceral adiposity and the bad fat depots that exist in our body, have a significant anti-inflammatory effect. And if you look at the data from the STEP trials that were done, looking at weight loss effects of semaglutide, a commonly used GLP-1 inhibitor, there was a good 30%-40% reduction in CRP levels, and that suggests that there is a significant anti-inflammatory effect. Now, whether it's a direct effect on anti-inflammatory pathways or is related to weight loss is a matter of debate. Piña: Are you using GLP-1 inhibitors in your HFpEF clinic? 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